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8/10
COA ✓Ships: USCredit/debit card
$58.47$64.97−10% · code PEPTIDEPRICES10mg package$58.47Checked aug 2, 2026Aug 2, 2026Buy Thymulin from BioLongevity Labs

Research details

Thymulin — research data

Half-lifeVery short (on the order of minutes). Continuous or frequent dosing models are used in research.
Regulatory statusNot FDA-approved
NCAA D1Unclear

Overview

About Thymulin

Mechanism of action

Nonapeptide thymic hormone secreted by thymic epithelial cells; its biological activity is strictly zinc-dependent (zinc must be bound for the peptide to be active). Promotes T-cell differentiation and NK-cell development and helps balance cytokine signaling; in animal models it sensitizes end-organs to hormone signaling (e.g. greater target-hormone output when combined with ACTH or hCG). Falling thymulin activity is among the earliest measurable markers of zinc deficiency, before serum or red-cell zinc drop.

Safety profile

Human safety data is sparse. Activity is strictly zinc-dependent, so effects are inseparable from zinc status. Generally low reported toxicity in animal models, but it should be treated as experimental. Research-use only; not for human consumption. · Endogenous zinc-dependent thymic nonapeptide (FTS); low toxicity in animal models and a benign physiologic profile, but human safety/PK data is sparse and there is no established dose. Activity is inseparable from zinc status; treat as experimental.

Related pages

More on Thymulin

Research

Studies & key findings

  • Thymulin is a zinc-dependent nonapeptide thymic hormone (also called FTS, facteur thymique sérique). Its biological activity REQUIRES bound zinc — without zinc the peptide is inactive — and declining thymulin activity is one of the earliest measurable markers of zinc deficiency, appearing before serum or red-cell zinc fall.
  • It promotes T-cell differentiation and NK-cell development and helps balance cytokine signaling. In animal models it 'sensitizes' end-organs to hormone signaling (e.g. greater target-hormone output when paired with ACTH or hCG), forming a link between immune status and the endocrine system.

4 peer-reviewed sources cited — clinical, preclinical, and regulatory.

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